Ecological Formulas - Allithiamine 50 mg 60 caps

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I was able to wipe out Candida Krusei but I do still have yeast overgrowth (white toungue) due to IBS-C. I had elevated oxalates but not sure if I still have it. Would taking calcium citrate help breakdown oxalate foods? It’s been extremely difficult eradicating my yeast infections. Dr. Lonsdale & Dr. Marrs, I was diagnosed with Parkinson’s last year, Symptoms began in 2018. I and 3 of my 4 siblings plus my mother are hypothyroid. I have been taking levothyroxine for over 10 years. I began taking thiamine HCL following Dr. Constantini’s protocol at the end of July 2021 hoping to improve my PD. I knew enough about the water soluble vitamins to know I needed them. I knew I urinated more than normal and so on. So I was taking vitamins and minerals but I was not entirely consistent in use. Arreola R., Quintero-Fabián S., López-Roa R.I., Flores-Gutiérrez E.O., Reyes-Grajeda J.P., Carrera-Quintanar L., Ortuño-Sahagún D. Immunomodulation and anti-inflammatory effects of garlic compounds. J. Immunol. Res. 2015;2015:401630. doi: 10.1155/2015/401630. My mother’s cancer doctor mentioned that it sounded like I had a major b12 deficiency and that I should try supplements. I did and it worked for a time, giving me improvements, but there was still residual numbness. Which gradually got worse.

So you are saying that Allithiamine or Thiamax would work as well or almost as well taken orally as Lipothiamine?. ( I was given pause because I had read that stomach acid could degrade part or all of the non enterically coated forms of TTFD.) I think I’ll increase my daily allithiamine from one capsule to two for a while, to build up my thiamine stores. My sibling was recently diagnosed with Parkinson’s (PD), and some people with PD are using thiamine to reduce their symptoms. Hopefully keeping my level high might reduce my risk of developing PD. I performed animal and clinical studies with thiamine tetrahydrofurfuryl (TTFD) for many years and found it to be an extremely valuable therapeutic nutrient. Any disease where energy deficiency is the underlying cause may respond to TTFD, unless permanent damage has accrued. Dr. Marrs and I believe that energy deficiency applies to any naturally occurring disease, even when a gene is at fault. For example, Japanese investigators found that TTFD protected mice from cyanide and carbon tetrachloride poisoning, an effect that was not shown by ordinary thiamine (Fujiwara, M. Absorption, excretion and fatal thiamine and its derivatives in the human body. In Shimazono, N, Katsura, E, eds. Beriberi and Thiamine. (pp 120-121) Tokyo, Igaku Shoin Ltd. 1965). They exposed a segment of dog’s intestine, disconnected it from its nerve supply and found that one of the disulfide derivatives stimulated peristalsis (the wavelike movement of the intestine). It is more than likely that TTFD could be used safely in patients with post operative paralysis of the intestine (paralytic ileus). Other Derivatives

The Japanese investigators synthesized a whole series of thiamine derivatives where the prosthetic group was attached to the carbon atom (bottom right C on the thiazole ring). They are all so-called open ring derivatives but the prosthetic group has to be separated by an enzyme in the body for the thiazole ring to close. The best known of these is known as Benfotiamine and several papers have been published concerning its benefits in the treatment of neuropathy. It has also been published that it does not cross into the brain, whereas TTFD does and this seems to be the major difference between Benfotiamine and Lipothiamine. Benfotiamine, a synthetic S-acyl thiamine derivative, has different mechanisms of action and a different pharmacological profile than lipid-soluble thiamine disulfide derivatives. It is predictable that TTFD would be the best choice since it has beneficial effects both inside and outside the brain and it certainly needs to be explored and researched further as a very valuable therapeutic agent. Thiamine Salts This advice about thiamine supplementation has given me hope that perhaps I may find some level of improvement to my failing health. I was always a fit, slim and healthy person before Lupron and its effects have been utterly devastating, mentally and physically. I literally feel that nobody, even my family and friends believe what I have gone through. It has been a very lonely experience. This “fat solubility” is extremely important because dietary thiamine has to be attached to a genetically determined protein, known as a transporter, to gain entry to cells. There are known to be diseases where the transporter is missing. Affected individuals have thiamine deficiency that does not respond to ordinary thiamine and are usually misdiagnosed. Therefore, a disulfide derivative that does not need the transporter is a method by which thiamine can be introduced to the cell when the transporter is missing. There is no difference between allithiamine and thiamine from a biological activity standpoint. It is this ability to pass the active vitamin through the cell membrane into the cell that provides the advantage. I have read a couple articles, including the comments, and I am fascinated by the subject, and your input. How much magnesium taurate–and when/how often? [He’s been taking magnesium citrate gummies very well for about a week, so if mag citrate is an acceptable option, that would be helpful.]

When I first came across all this info on thiamine I started supplementing and the worst of my mental confusion, nausea, loss of appetite went away within a few days. I stopped the B1 because of gut pain and the symptoms increased again, I then restarted the B1 and they decreased again. (Unfortunately I had been supplementing for a bit before performing the test but intermittently) I am now using the Authia cream since I struggle with oral supplementation. I have noticed that I feel really tired after applying the cream for most of the day. I seem to remember reading that this would also suggest deficiency as otherwise supplementing would have no effect. I would love to know what caused my health problems, but my tests have mostly been normal except for Graves’ disease. Even my genome looks okay, with no problems on the thiamine or cobalamin transporter genes. It makes me a bit uneasy to keep my B12 level so high, so I’d really like to understand why I need it.Would upping the dose orally on Allthiamine theoretically compensate for any alcohol induced issues? Neither physician is sure. In the case of my friend, he went from a strong suspicion to feeling it’s just a possibility after thinking it through. He didn’t examine me, it was a phone conversation. He now feels that it may just be b1 deficiency considering the alcohol use was roughly less than a year and I had stopped for 3 months before and was asymptomatic those 3 months , and just presented symptoms after 2 weeks of returning to moderate consumption. I’m wondering if you’ve ever seen anyone who had symptoms like mine and if this can all just be chalked up to a b1 deficiency. That maybe I’ll be able to eat something other than meat in the future. I gave thiamine a try (Thiamine Mononitrate) 50 mg but after a couple of days, I had some bad reaction to the product (tachycardia, worst dysautonomia upon standing). I was surprised because the dose is fairly minimal compared to those of other patients (some take north of 1g). I was wondering if this is the kind of paradox you have seen in the past? Do you think it is due to the mononitrate part of the product or thiamine itself? Should I try thiamine HCL instead? Jang H.-J., Lee H.-J., Yoon D.-K., Ji D.-S., Kim J.-H., Lee C.-H. Antioxidant and antimicrobial activities of fresh garlic and aged garlic by-products extracted with different solvents. Food Sci. Biotechnol. 2017;27:219–225. doi: 10.1007/s10068-017-0246-4.